Further case of paroxysmal exercise‐induced dystonia and some insights into pathogenesis
Identifieur interne : 004449 ( Main/Exploration ); précédent : 004448; suivant : 004450Further case of paroxysmal exercise‐induced dystonia and some insights into pathogenesis
Auteurs : Michael H. Barnett [Royaume-Uni] ; Paul R. Jarman [Royaume-Uni] ; Simon J. R. Heales [Royaume-Uni] ; Kailash P. Bhatia [Royaume-Uni]Source :
- Movement Disorders [ 0885-3185 ] ; 2002-11.
Descripteurs français
- Pascal (Inist)
- Wicri :
- topic : Homme.
English descriptors
- KwdEn :
- Adult, Case study, Cerebrospinal fluid, Dopamine (physiology), Dystonia, Dystonia (diagnosis), Dystonia (physiopathology), Exercise (physiology), Homovanillic Acid (cerebrospinal fluid), Human, Humans, Hydroxyindoleacetic Acid (cerebrospinal fluid), Male, Neurologic Examination, Pathogenesis, Physical exercise, Synaptic Transmission (physiology), Triggering, Video recording, cerebrospinal fluid, dopamine, homovanillic acid, hydroxyindoleacetic acid, paroxysmal exercise‐induced dystonia.
- MESH :
- chemical , cerebrospinal fluid : Homovanillic Acid, Hydroxyindoleacetic Acid.
- chemical , physiology : Dopamine.
- diagnosis : Dystonia.
- physiology : Exercise, Synaptic Transmission.
- physiopathology : Dystonia.
- Adult, Humans, Male, Neurologic Examination.
Abstract
Cerebrospinal fluid (CSF) analysis of pterin and monamine metabolites was performed before and after an attack in a patient with paroxysmal exercise‐induced dystonia. A twofold increase in CSF homovanillic acid and 5‐hydroxyindoleacetic acid after an attack was measured. This finding lends support to the hypothesis that increased dopaminergic transmission contributes to the clinical features of the hyperkinetic movement disorders. © 2002 Movement Disorder Society
Url:
DOI: 10.1002/mds.10291
Affiliations:
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Le document en format XML
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Disord.</title><idno type="ISSN">0885-3185</idno><idno type="eISSN">1531-8257</idno><imprint><publisher>Wiley Subscription Services, Inc., A Wiley Company</publisher><pubPlace>New York</pubPlace><date type="published" when="2002-11">2002-11</date><biblScope unit="vol">17</biblScope><biblScope unit="issue">6</biblScope><biblScope unit="page" from="1386">1386</biblScope><biblScope unit="page" to="1387">1387</biblScope></imprint><idno type="ISSN">0885-3185</idno></series><idno type="istex">B6C200B63011197A343702941365820C9C753E7C</idno><idno type="DOI">10.1002/mds.10291</idno><idno type="ArticleID">MDS10291</idno></biblStruct></sourceDesc><seriesStmt><idno type="ISSN">0885-3185</idno></seriesStmt></fileDesc><profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Adult</term><term>Case study</term><term>Cerebrospinal fluid</term><term>Dopamine (physiology)</term><term>Dystonia</term><term>Dystonia (diagnosis)</term><term>Dystonia (physiopathology)</term><term>Exercise (physiology)</term><term>Homovanillic Acid (cerebrospinal fluid)</term><term>Human</term><term>Humans</term><term>Hydroxyindoleacetic Acid (cerebrospinal fluid)</term><term>Male</term><term>Neurologic Examination</term><term>Pathogenesis</term><term>Physical exercise</term><term>Synaptic Transmission (physiology)</term><term>Triggering</term><term>Video recording</term><term>cerebrospinal fluid</term><term>dopamine</term><term>homovanillic acid</term><term>hydroxyindoleacetic acid</term><term>paroxysmal exercise‐induced dystonia</term></keywords><keywords scheme="MESH" type="chemical" qualifier="cerebrospinal fluid" xml:lang="en"><term>Homovanillic Acid</term><term>Hydroxyindoleacetic Acid</term></keywords><keywords scheme="MESH" type="chemical" qualifier="physiology" xml:lang="en"><term>Dopamine</term></keywords><keywords scheme="MESH" qualifier="diagnosis" xml:lang="en"><term>Dystonia</term></keywords><keywords scheme="MESH" qualifier="physiology" xml:lang="en"><term>Exercise</term><term>Synaptic Transmission</term></keywords><keywords scheme="MESH" qualifier="physiopathology" xml:lang="en"><term>Dystonia</term></keywords><keywords scheme="MESH" xml:lang="en"><term>Adult</term><term>Humans</term><term>Male</term><term>Neurologic Examination</term></keywords><keywords scheme="Pascal" xml:lang="fr"><term>Acide homovanillique</term><term>Acide hydroxyindole-acétique</term><term>Dystonie</term><term>Déclenchement</term><term>Enregistrement vidéo</term><term>Etude cas</term><term>Exercice physique</term><term>Homme</term><term>Liquide céphalorachidien</term><term>Pathogénie</term></keywords><keywords scheme="Wicri" type="topic" xml:lang="fr"><term>Homme</term></keywords></textClass><langUsage><language ident="en">en</language></langUsage></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">Cerebrospinal fluid (CSF) analysis of pterin and monamine metabolites was performed before and after an attack in a patient with paroxysmal exercise‐induced dystonia. A twofold increase in CSF homovanillic acid and 5‐hydroxyindoleacetic acid after an attack was measured. This finding lends support to the hypothesis that increased dopaminergic transmission contributes to the clinical features of the hyperkinetic movement disorders. © 2002 Movement Disorder Society</div></front></TEI><affiliations><list><country><li>Royaume-Uni</li></country><region><li>Angleterre</li><li>Grand Londres</li></region><settlement><li>Londres</li></settlement></list><tree><country name="Royaume-Uni"><region name="Angleterre"><name sortKey="Barnett, Michael H" sort="Barnett, Michael H" uniqKey="Barnett M" first="Michael H." last="Barnett">Michael H. Barnett</name></region><name sortKey="Bhatia, Kailash P" sort="Bhatia, Kailash P" uniqKey="Bhatia K" first="Kailash P." last="Bhatia">Kailash P. Bhatia</name><name sortKey="Heales, Simon J R" sort="Heales, Simon J R" uniqKey="Heales S" first="Simon J. R." last="Heales">Simon J. R. Heales</name><name sortKey="Jarman, Paul R" sort="Jarman, Paul R" uniqKey="Jarman P" first="Paul R." last="Jarman">Paul R. Jarman</name></country></tree></affiliations></record>Pour manipuler ce document sous Unix (Dilib)
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